logo

670 nm Light Therapy to Protect vs Photoreceptor Cell Death

PDF Publication Title:

670 nm Light Therapy to Protect vs Photoreceptor Cell Death ( 670-nm-light-therapy-protect-vs-photoreceptor-cell-death )

Previous Page View | Next Page View | Return to Search List

Text from PDF Page: 012

International Journal of Photoenergy 11 [22] R. Albarracin, J. Eells, and K. Valter, “Photobiomodulation protects the retina from light-induced photoreceptor degener- ation,” Investigative Ophthalmology and Visual Science, vol. 52, no. 6, pp. 3582–3592, 2011. [23] R. Albarracin and K. Valter, “670 nm red light preconditioning supports Mu ̈ller cell function: evidence from the white light- induced damage model in the rat retina,” Photochemistry and Photobiology, vol. 88, no. 6, pp. 1418–1427, 2012. [24] R. S. Albarracin and K. Valter, “Treatment with 670-nm light protects the cone photoreceptors from white light-induced degeneration,” Advances in Experimental Medicine and Biology, vol. 723, pp. 121–128, 2012. [25] C. Qu, W. Cao, Y. Fan, and Y. Lin, “Near-infrared light protect the photoreceptor from light-induced damage in rats,” Advances in Experimental Medicine and Biology, vol. 664, pp. 365–374, 2010. [26] M.Rutar,R.Natoli,R.Albarracin,K.Valter,andJ.Provis,“670- nm light treatment reduces complement propagation following retinal degeneration,” Journal of Neuroinflammation, vol. 9, article 257, 2012. [27] T. Karu, “Primary and secondary mechanisms of action of visible to near-IR radiation on cells,” Journal of Photochemistry and Photobiology B: Biology, vol. 49, no. 1, pp. 1–17, 1999. [28] M. T. T. Wong-Riley, H. L. Liang, J. T. Eells et al., “Photo- biomodulation directly benefits primary neurons functionally inactivated by toxins: role of cytochrome c oxidase,” The Journal of Biological Chemistry, vol. 280, no. 6, pp. 4761–4771, 2005. [29] R. Begum, M. B. Powner, N. Hudson, C. Hogg, and G. Jeffery, “Treatment with 670 nm light up regulates cytochrome C oxidase expression and reduces inflammation in an age-related macular degeneration model,” PLoS ONE, vol. 8, no. 2, Article ID e57828, 2013. [30] M. T. T. Wong-Riley, X. Bai, E. Buchmann, and H. T. Whelan, “Light-emitting diode treatment reverses the effect of TTX on cytochrome oxidase in neurons,” NeuroReport, vol. 12, no. 14, pp. 3033–3037, 2001. [31] T. I. Karu, “Multiple roles of cytochrome c oxidase in mam- malian cells under action of red and IR-A radiation,” IUBMB Life, vol. 62, no. 8, pp. 607–610, 2010. [32] M. Rutar, J. M. Provis, and K. Valter, “Brief exposure to damaging light causes focal recruitment of macrophages, and long-term destabilization of photoreceptors in the albino rat retina,” Current Eye Research, vol. 35, no. 7, pp. 631–643, 2010. [33] R. Albarracin, R. Natoli, M. Rutar, K. Valter, and J. Pro- vis, “670nm light mitigates oxygen-induced degeneration in C57BL/6J mouse retina,” BMC Neuroscience, vol. 14, article 125, 2013. [34] R. Natoli, Y. Zhu, K. Valter, S. Bisti, J. Eells, and J. Stone, “Gene and noncoding RNA regulation underlying photoreceptor pro- tection: microarray study of dietary antioxidant saffron and photobiomodulation in rat retina,” Molecular Vision, vol. 16, pp. 1801–1822, 2010. [35] J. T. Eells, M. T. T. Wong-Riley, J. VerHoeve et al., “Mito- chondrial signal transduction in accelerated wound and retinal healing by near-infrared light therapy,” Mitochondrion, vol. 4, no. 5-6, pp. 559–567, 2004. [36] E. J. Calabrese, “Converging concepts: adaptive response, pre- conditioning, and the Yerkes-Dodson Law are manifestations of hormesis,” Ageing Research Reviews, vol. 7, no. 1, pp. 8–20, 2008. [37] M. P. Mattson, “Hormesis defined,” Ageing Research Reviews, vol. 7, no. 1, pp. 1–7, 2008. [38] S. Wu, F. Zhou, Y. Wei, W. R. Chen, Q. Chen, and D. Xing, “Cancer phototherapy via selective photoinactivation of respi- ratory chain oxidase to trigger a fatal superoxide anion burst,” Antioxidants and Redox Signaling, vol. 20, no. 5, pp. 733–746, 2014. [39] S. Wu, D. Xing, X. Gao, and W. R. Chen, “High fluence low- power laser irradiation induces mitochondrial permeability transition mediated by reactive oxygen species,” Journal of Cellular Physiology, vol. 218, no. 3, pp. 603–611, 2009. [40] I. Kokkinopoulos, A. Colman, C. Hogg, J. Heckenlively, and G. Jeffery, “Age-related retinal inflammation is reduced by 670 nm light via increased mitochondrial membrane potential,” Neurobiology of Aging, vol. 34, no. 2, pp. 602–609, 2013. [41] D. Pastore, M. Greco, and S. Passarella, “Specific helium- neon laser sensitivity of the purified cytochrome c oxidase,” International Journal of Radiation Biology, vol. 76, no. 6, pp. 863–870, 2000. [42] C. Desler, T. L. Hansen, J. B. Frederiksen, M. L. Marcker, K. K. Singh, and L. Juel Rasmussen, “Is there a link between mitochondrial reserve respiratory capacity and aging?” Journal of Aging Research, vol. 2012, Article ID 192503, 9 pages, 2012. [43] B.E.Sansbury,S.P.Jones,D.W.Riggs,V.M.Darley-Usmar,and B. G. Hill, “Bioenergetic function in cardiovascular cells: the importanceofthereservecapacityanditsbiologicalregulation,” Chemico-Biological Interactions, vol. 191, no. 1–3, pp. 288–295, 2011. [44] D. G. Nicholls, “Oxidative stress and energy crises in neuronal dysfunction,” Annals of the New York Academy of Sciences, vol. 1147, pp. 53–60, 2008. [45] N.YadavaandD.G.Nicholls,“Sparerespiratorycapacityrather than oxidative stress regulates glutamate excitotoxicity after partial respiratory inhibition of mitochondrial complex I with rotenone,” Journal of Neuroscience, vol. 27, no. 27, pp. 7310–7317, 2007. [46] B. G. Hill, A. N. Higdon, B. P. Dranka, and V. M. Darley- Usmar, “Regulation of vascular smooth muscle cell bioenergetic function by protein glutathiolation,” Biochimica et Biophysica Acta (BBA)—Bioenergetics, vol. 1797, no. 2, pp. 285–295, 2010. [47] T. I. Karu, L. V. Pyatibrat, and N. I. Afanasyeva, “A novel mitochondrial signaling pathway activated by visible-to-near infrared radiation,” Photochemistry and Photobiology, vol. 80, no. 2, pp. 366–372, 2004. [48] M. D. Brand and D. G. Nicholls, “Assessing mitochondrial dysfunction in cells,” Biochemical Journal, vol. 435, no. 2, pp. 297–312, 2011. [49] G. Villani and G. Attardi, “In vivo control of respiration by cytochrome c oxidase in human cells,” Free Radical Biology and Medicine, vol. 29, no. 3-4, pp. 202–210, 2000. [50] G. C. Brown, “Regulation of mitochondrial respiration by nitric oxide inhibition of cytochrome c oxidase,” Biochimica et Biophysica Acta, vol. 1504, no. 1, pp. 46–57, 2001. [51] G. C. Brown and C. E. Cooper, “Nanomolar concentrations of nitric oxide reversibly inhibit synaptosomal respiration by competing with oxygen at cytochrome oxidase,” FEBS Letters, vol. 356, no. 2-3, pp. 295–298, 1994. [52] M.W.J.Cleeter,J.M.Cooper,V.M.Darley-Usmar,S.Moncada, and A. H. V. Schapira, “Reversible inhibition of cytochrome c oxidase, the terminal enzyme of the mitochondrial respiratory chain, by nitric oxide. Implications for neurodegenerative diseases,” FEBS Letters, vol. 345, no. 1, pp. 50–54, 1994. [53] G. C. Brown and V. Borutaite, “Nitric oxide, mitochondria, and cell death,” IUBMB Life, vol. 52, no. 3–5, pp. 189–195, 2001.

PDF Image | 670 nm Light Therapy to Protect vs Photoreceptor Cell Death

670-nm-light-therapy-protect-vs-photoreceptor-cell-death-012

PDF Search Title:

670 nm Light Therapy to Protect vs Photoreceptor Cell Death

Original File Name Searched:

Efficacy_of_670_nm_Light_Therapy_to_Protect_agains.pdf

DIY PDF Search: Google It | Yahoo | Bing

Cruise Ship Reviews | Luxury Resort | Jet | Yacht | and Travel Tech More Info

Cruising Review Topics and Articles More Info

Software based on Filemaker for the travel industry More Info

The Burgenstock Resort: Reviews on CruisingReview website... More Info

Resort Reviews: World Class resorts... More Info

The Riffelalp Resort: Reviews on CruisingReview website... More Info

CONTACT TEL: 608-238-6001 Email: greg@cruisingreview.com | RSS | AMP